Thursday, January 2, 2020
The man to discover Alzheimerââ¬â¢s disease was Alois...
The man to discover Alzheimerââ¬â¢s disease was Alois Alzheimer; he discovered it after a patient with an abnormal mental illness passed. Alzheimerââ¬â¢s, also known as AD, is named after Alois Alzheimer and is a continuing loss of brain function that affects thinking skills, such as forming or retrieving memories and judgment. I think that it took him so long to discover that it was a new disease because Alzheimerââ¬â¢s can be so subtle that only a drastic change in the view of the brain can show the outcome of the cells. This disease affects older people however it is not a normal part of aging. AD does not have a cure yet, scientists are however trying to find the root of the disease and control its destructive powers. Currently there is no cureâ⬠¦show more contentâ⬠¦There is usually a way to find out if you have Alzheimerââ¬â¢s, this could be determined either through thorough examination of the cells or understanding of the genetic history of the family. There are some potential treatments for Alzheimerââ¬â¢s that scientists have contemplated such as Zinc and the prevention of a C1q protein. Zinc is critical for the brain and without it neurons can die. As a possible treatment remedy, Zinc can be added to a diet. Also Zn can decline the amount of Amyloid plaques. Recently, Ben Barres who studied at Stanford University School of Medicine conducted a study which involves a protein called C1q. C1q is deemed important because it builds up around the brain where the nerve cells are, this is important because the destruction of nerve cells leads to dementia. C1q is responsible for the elimination of hazardous material and is usually not found in the brain. A study was conducted with the development of an infants brain stating that the growth of synapsis was too quick and there were an abundance of them and something had to get rid of unnecessary synapsis causing a dangerous amount of C1q. Barres is currently working on a possible drug t hat could prevent the C1q protein. With the current curiosity that AD brings on people, I believe that in no time we will have a treatment. My interest in Alzheimerââ¬â¢s stems of the effects it has on an individual and
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